Nitric oxide modulation of the hypothalamo-neurohypophyseal system
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Nitric oxide modulation of the hypothalamo-neurohypophyseal system.
Nitric oxide (NO), a free radical gas produced endogenously from the amino acid L-arginine by NO synthase (NOS), has important functions in modulating vasopressin and oxytocin secretion from the hypothalamo-neurohypophyseal system. NO production is stimulated during increased functional activity of magnocellular neurons, in parallel with plastic changes of the supraoptic nucleus (SON) and parav...
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Endothelin (ET) peptides stimulate vasopressin (AVP) secretion via ET(B) receptors at hypothalamic loci. Nitric oxide modulates the actions of ET in the cardiovascular system and also influences neurotransmission and specifically suppresses firing of magnocellular neurons. The purpose of these studies was to ascertain whether nitric oxide, generated in response to ET(B) receptor stimulation, bu...
متن کاملCALL FOR PAPERS Neurohypophyseal Hormones: From Genomics and Physiology to Disease Nitric oxide modulation of ETB receptor-induced vasopressin release by rat and mouse hypothalamo-neurohypophyseal explants
Rossi, Noreen F., and William H. Beierwaltes. Nitric oxide modulation of ETB receptor-induced vasopressin release by rat and mouse hypothalamo-neurohypophyseal explants. Am J Physiol Regul Integr Comp Physiol 290: R1208–R1215, 2006. First published December 15, 2005; doi:10.1152/ajpregu.00701.2005.—Endothelin (ET) peptides stimulate vasopressin (AVP) secretion via ETB receptors at hypothalamic ...
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The hypothalamo-neurohypophyseal system (HNS) mediates neuroendocrine responses to dehydration through the action of the antidiuretic hormone vasopressin (VP). VP is synthesized as part of a prepropeptide in magnocellular neurons of the hypothalamic supraoptic nucleus (SON) and paraventricular nucleus. This precursor is processed during transport to axon terminals in the posterior pituitary gla...
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Endothelin (ET) peptides stimulate AVP secretion via ETB receptors at hypothalamic loci. Nitric oxide modulates the actions of ET in the cardiovascular system and also influences neurotransmission and specifically suppresses firing of magnocellular neurons. The purpose of these studies was to ascertain whether nitric oxide, generated in response to ETB receptor stimulation, buffers the stimulat...
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ژورنال
عنوان ژورنال: Brazilian Journal of Medical and Biological Research
سال: 2004
ISSN: 0100-879X
DOI: 10.1590/s0100-879x2004000400001